Digoxin inhibits a Na⁺-K⁺ ATPase pump in the membranes of the heart cells (myocytes). This is the causal agent of an increase in the level of sodium ions in the myocytes, which then leads to the rise in the level of calcium ions. This increased level of ca increases a contractility of the myocardium (muscle of the heart).

Digoxin as well lessens a conductivity of electrical impulses through the AV node, making it a normally utilized drug within controlling a heart rate during atrial fibrillation or atrial flutter.

Its utilize within congestive heart failure during sinus rhythm is controversial.

A select few physical properties of lanoxin come a water supply solubility of 64.8mg/L at Twenty-five°C & melting point at 249°C.